While there is still no research on how greater potency affects the long-term risks of marijuana use, a higher amount of THC is likely to lead to higher rates of dependence and. There are some risks and side effects associated with both short-term and long-term cannabis use. Short-term effects include impaired short-term memory, motor coordination (impaired driving ability), impaired judgment and, when consumed in high doses, paranoia and psychosis. Prolonged or intensive use is associated with altered brain development, especially among those who use cannabis in adolescence.
It can also increase the risk of developing chronic psychosis disorders, such as schizophrenia, among predisposed people. Unlike alcohol, which is metabolized at a relatively constant rate, THC is metabolized at an exponentially decreasing rate, since the level of THC in the blood first drops rapidly, followed by a slower decline as lower levels of THC are reached. The higher the amount of tetrahydrocannabinol (THC) in marijuana (in other words, concentration or concentration), the stronger the effects that marijuana can have on the brain. While there is still no research on how greater potency affects the long-term risks of marijuana use, more THC is likely to cause higher rates of dependency and addiction.
THCA also refers to a different molecule that is a precursor to THC, which has caused confusion among lawyers who read toxicology reports that refer to THCA. Marijuana is a psychoactive drug that contains nearly 500 chemicals, including THC, a mind-altering compound that causes harmful health effects. The maximum level of THC can occur when measuring low deterioration, and high deterioration can be measured when the THC level is low. This low concentration of THC in the blood is not sufficient to cause the high concentration in the brain needed for deterioration.
THC is psychotropic and is capable of producing a “high”, while CBD is not psychotropic and does not exert this effect. The State Crime Laboratory reports refer to 11-nor-delta-9-tetrahydrocannabinol-9-carboxylic acid with the abbreviation THCA. Cannabidiol synthase (CBDA synthase) and tetrahydrocannabinolic acid synthase (THCA synthase) catalyze the oxidative cyclization of CBGA into CBDA and THCA, respectively. The blood concentration decreases rapidly after you stop smoking, as no more THC enters the blood and what is left in the blood is distributed to adipose tissue.
It is not recommended to try to predict effects based solely on blood THC concentrations, and it is currently impossible to predict specific effects based on THC-COOH concentrations.